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Endogenous TSH causes stimulation of [14C]acetate incorporation into phosphatidylcholine in cultures of thyroid follicular epithelium. The maximal effect on phosphatidylcholine synthesis is obtained at a level of endogenous TSH that is within the normal range of serum concentrations found in the human. The effects of thyroxine or triiodothyronine on phosphatidylcholine synthesis were also examined. In contrast to TSH, thyroxine had no effect on phosphatidylcholine synthesis, and triiodothyronine was slightly inhibitory. Organ culture studies have also been performed on the other thyroid hormone, 3, 5-dimethyl-3, 5-dihydroxybenzoic acid, which we recently discovered to stimulate thyroid function in the rat. Again, phosphatidylcholine synthesis was stimulated by TSH, but not by 3, 5-dimethyl-3, 5-dihydroxybenzoic acid. It is concluded that phosphatidylcholine synthesis is a direct action of TSH on the thyroid gland. size of the tumour, high AFP and total bilirubin levels, as well as the size of the tumour, as were the presence of nodal metastases, high AFP and bilirubin levels, were significantly related to shorter OS. Our data are more powerful, since they show a relationship between aggressive factors and shorter OS.
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